Tag Archives: AmpC

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Objetivo general:

Explicar los principales mecanismos de resistencia en bacilos Gram negativos de relevancia clínica.

Objetivos específicos:

1. Describir los mecanismos de resistencia frente a betalactámicos en especies de Enterobacterales.


2. Explicar los mecanismos de resistencia a aminoglucósidos y fluoroquinolonas en Enterobacterales.


3. Identificar los mecanismos de resistencia a betalactámicos en Pseudomonas aeruginosa .


4. Resumir los métodos fenotípicos de detección de los principales mecanismos de resistencia en Enterobacterales y P.aeruginosa .

Duración: 8 semanas.

Carga horaria : 30 horas de estudio.

 

Facilitadora: Lcda. Carolina Macero

Bacteriólogo clínico.

 

¿Qué aprenderás con esta Guía de Micro?

• Aspectos microbiológicos a considerar de las especies Enterobacterales con mayor importancia clínica, en la resistencia frente a los principales antibacterianos.


• Mecanismos de resistencia a betalactámicos, aminoglucósidos y fluoroquinolonas en Enterobacterales de impacto clínico.


• Aspectos microbiológicos de Pseudomonas aeruginosa en la resistencia a los principales antibacterianos.


• Mecanismos de resistencia frente a betalactámicos en Pseudomonas aeruginosa .


• Métodos fenotípicos utilizados en la detección de los principales mecanismos de resistencia en las especies de Enterobacterales más frecuentes y P.aeruginosa .

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Desarrollo de un algoritmo para discriminar entre la producción de β-lactamasa AmpC mediada por plásmidos y cromosomas en Escherichia coli, mediante una caracterización fenotípica y genotípica.

[/nectar_highlighted_text][vc_column_text]Para fines de control de infecciones, es importante poder discriminar entre la producción de AmpC mediada por plásmidos (pAmpC) y la hiperproducción de AmpC mediada por cromosomas (cAmpC) en bacterias Gram-negativas, ya que pAmpC requiere precauciones de aislamiento para minimizar el riesgo de transmisión horizontal de genes. La detección de pAmpC en Escherichia coli es un desafío, ya que tanto la producción de pAmpC como la hiperproducción de cAmpC pueden conducir a una resistencia a las cefalosporinas de tercera generación.

Este artículo presenta un algoritmo pragmático para diferenciar genotipos ampC en E. coli basado en pruebas de susceptibilidad fenotípica.[/vc_column_text][text-with-icon icon_type="font_icon" icon="icon-book" color="Accent-Color"]

Journal of Antimicrobial Chemotherapy  , volumen 74, número 12, diciembre de 2019, páginas 3481–3488, https://doi.org/10.1093/jac/dkz362

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